Skip to content
AkademScholar

Products

For developers

AkademBasesoonDeveloper API
Journal

Frontiers in Cell and Developmental Biology

ISSN:
2296-634X
2
h-index
16
Citations
4
Works

Trust signals

1 of 3 signals present

Independent positive signals — not a single “predatory/legitimate” verdict, but a converging picture.

  • DOAJ
    No data
    doaj.org
  • OAK list
    No data
    OAK
  • Valid ISSN
    Present
    ISSN

This is an information label, not an official ruling. A missing signal is not a penalty — a regional journal may simply not be in DOAJ.

Trends over time

Performance over time

Citation overview

Publications (bars) and citations (line) by year

  • Publications
  • Citations
013450358102021202220232024202520262021: Publications 3, Citations 62022: Publications 0, Citations 22023: Publications 1, Citations 32024: Publications 0, Citations 12025: Publications 0, Citations 32026: Publications 0, Citations 1

Citation history

0358102021202220232024202520262021: Citations 62022: Citations 22023: Citations 32024: Citations 12025: Citations 32026: Citations 1

Publication history

013452021202220232024202520262021: Publications 32022: Publications 02023: Publications 12024: Publications 02025: Publications 02026: Publications 0

h-index evolution

Cumulative h-index by year

0122021202220232024202520262021: h-index 22022: h-index 22023: h-index 22024: h-index 22025: h-index 22026: h-index 2

Most-cited works

  1. Cell Death Induction and Protection by Activation of Ubiquitously Expressed Anion/Cation Channels. Part 1: Roles of VSOR/VRAC in Cell Volume Regulation, Release of Double-Edged Signals and Apoptotic/Necrotic Cell Death20219 citations
  2. Cell Death Induction and Protection by Activation of Ubiquitously Expressed Anion/Cation Channels. Part 2: Functional and Molecular Properties of ASOR/PAC Channels and Their Roles in Cell Volume Dysregulation and Acidotoxic Cell Death20215 citations
  3. Positive Inotropic Effects of ATP Released via the Maxi-Anion Channel in Langendorff-Perfused Mouse Hearts Subjected to Ischemia-Reperfusion20212 citations
  4. Cell death induction and protection by activation of ubiquitously expressed anion/cation channels. Part 3: the roles and properties of TRPM2 and TRPM720230 citations
View in AkademIndex →