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Journal

BIOPHYSICS

ISSN:
0006-3509
1
h-index
1
Citations
7
Works
0.00
2-year mean citedness

Trust signals

1 of 3 signals present

Independent positive signals — not a single “predatory/legitimate” verdict, but a converging picture.

  • DOAJ
    No data
    doaj.org
  • OAK list
    No data
    OAK
  • Valid ISSN
    Present
    ISSN

This is an information label, not an official ruling. A missing signal is not a penalty — a regional journal may simply not be in DOAJ.

Trends over time

Performance over time

Citation overview

Publications (bars) and citations (line) by year

  • Publications
  • Citations
0120120092010201120132016201720252009: Publications 1, Citations 02010: Publications 1, Citations 02011: Publications 1, Citations 02013: Publications 1, Citations 02016: Publications 2, Citations 02017: Publications 0, Citations 12025: Publications 1, Citations 0

Citation history

0120092010201120132016201720252009: Citations 02010: Citations 02011: Citations 02013: Citations 02016: Citations 02017: Citations 12025: Citations 0

Publication history

01220092010201120132016201720252009: Publications 12010: Publications 12011: Publications 12013: Publications 12016: Publications 22017: Publications 02025: Publications 1

h-index evolution

Cumulative h-index by year

0120092010201120132016201720252009: h-index 02010: h-index 02011: h-index 02013: h-index 02016: h-index 02017: h-index 12025: h-index 1

Most-cited works

  1. The possible role of nonbilayer structures in regulating ATP synthase activity in mitochondrial membranes20161 citations
  2. Numerical study of the stability of some autowave solutions in a mathematical model of blood clotting20090 citations
  3. Fluorescence of acridine orange in the composition of blood and tissues20100 citations
  4. Quantum theory of the hydrogen key in DNA20110 citations
  5. Thermoadsorptive separation of DNA by size using a polymeric sorbent20130 citations
  6. On a possible mechanism of the effect of microwave radiation on biological macromolecules20160 citations
  7. Oxidized Disulfiram Derivatives Activate an Integrated Stress Response and Cause Paraptosis of Human Cancer Cells BT47420250 citations
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